Information report for RL9;SLL1
Gene Details
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Functional Descriptions
- RL9 is mainly expressed in roots, leaves, and flowers.
- The SLL1 gene was expressed at the lateral root tip, whereas SLL1 expression was not detected in the elongation zone of the crown roots.
- These results indicate that the lateral root specific defect in SLL1 mutant is caused by the different expression patterns of SLL1 in lateral and crown roots.
- In this study, we characterized two rice (Oryza sativa) allelic rolled-leaf mutants, rolled leaf 9-1 (RL9-1) and RL9-2, which display very similar phenotypes with completely adaxialized leaves and malformed spikelets.
- Here, we show the isolation and functional characterization of SHALLOT-LIKE1 (SLL1), a key gene controlling rice leaf rolling.
- SLL1 is transcribed in various tissues and accumulates in the abaxial epidermis throughout leaf development.
- Our findings identify the role of SLL1 in the modulation of leaf abaxial cell development and in sustaining abaxial characteristics during leaf development.
- An SLL1 mutant has decreased lateral root growth due to a defect in the cell elongation.
- SLL1 encodes a SHAQKYF class MYB family transcription factor belonging to the KANADI family.
- We have identified a gene, SHORT LATERAL ROOT LENGTH1 (SLL1), which is important for the elongation of lateral roots in rice.
- In addition, SLL1 over-expressers produced significantly longer lateral roots compared to the wild-type, and thus SLL1 gene would be very useful for improving rice root architecture.
- SLL1, which encodes a member of the stearoyl-acyl carrier protein fatty acid desaturase family, is involved in cell elongation in lateral roots via regulation of fatty acid content in rice.
- SLL1 deficiency leads to defective programmed cell death of abaxial mesophyll cells and suppresses the development of abaxial features.
- We measured the fatty acid content and found that the 18:0 content in the SLL1 mutant root was approximately 4 times that in the wild-type root.
- The transient expression of a RL9-GFP (green fluorescent protein) fusion protein has indicated that RL9 protein is localized in the nucleus, suggesting that RL9 acts as a putative transcription factor.
- Additionally, SLL1 deficiency results in increased chlorophyll and photosynthesis.
- AH2 encodes a MYB domain protein that determines hull fate and affects grain yield and quality in rice.
- In the study, AH2 encodes a MYB domain protein, and functions in the development of hull and grain.
- Mutation of AH2 produces smaller grains and alters grain quality including decreased amylose content and gel consistency, and increased protein content.
- Our results revealed that AH2 plays an important role in the determination of hull epidermis development, palea identity, and grain size.
- Meantime, part of the hull lost the outer silicified cells, and induces a transformation of the outer rough epidermis to inner smooth epidermis cells, and the body of the palea was reduced in the AH2 muant.
Functional Keywords
- root , crown-root , leaf , flower , spikelet , leaf-rolling , growth , transcription-factor , lateral-root , cell-death , photosynthesis , architecture , crown , cell-elongation , leaf-development , root-architecture , grain , development , grain-yield , yield , grain-size , palea , quality , epidermis , grain-quality
Literature and News
- SLL1, which encodes a member of the stearoyl-acyl carrier protein fatty acid desaturase family, is involved in cell elongation in lateral roots via regulation of fatty acid content in rice . DOI: 10.1016/j.plantsci.2013.01.003 ; PMID: 23602094
- SHALLOT-LIKE1 is a KANADI transcription factor that modulates rice leaf rolling by regulating leaf abaxial cell development . DOI: 10.1105/tpc.108.061457 ; PMID: 19304938
- Isolation and characterization of a rice mutant with narrow and rolled leaves . DOI: 10.1007/s00425-010-1180-3 ; PMID: 20443024
- ROLLED LEAF 9, encoding a GARP protein, regulates the leaf abaxial cell fate in rice . DOI: 10.1007/s11103-008-9365-x ; PMID: 18594992
- AH2 encodes a MYB domain protein that determines hull fate and affects grain yield and quality in rice . DOI: 10.1111/tpj.14481 ; PMID: 31357245
Gene Resources
- UniProt: Q0J235
- EMBL: AP005904, AP008215, AP014965
- AlphaFoldDB: Q0J235
- KEGG: dosa:Os09g0395300
- InterPro: IPR001005, IPR006447, IPR009057
- PANTHER: PTHR31496, PTHR31496:SF3
- SUPFAM: SSF46689
- Gene3D: 1.10.10.60
- SWISS-MODEL: Q0J235
- eggNOG: ENOG502QQZR
Sequences
cDNA Sequence
- >LOC_Os09g23200.1
ATGGCAATGGTGCGGGAGCTCGAGTTGATGACGAGCTGGAGCAACAGCATGGGGCGACACCGCTACCCAACGCGCATCCTCGTCGATTCCTTCGGGCATAAGTGCAGTGCCTCTGACAAGGGTGTGTGGACCTCCTGCAGCATTCGCGCACCCTTGCAAGGACGCGGCTCCTTCCGACGTGGCGCGAATATTCGCTTTGGGAGCTTGCCGAGCAGCGCGGCGGTGGCGACGTCGGGGGGAGGGCGTGGCGGCGGCGGGGTGGTGGTGGGAGGAGGAGGAGGGGACCCGTGGCGGAGGCTGGATGGGTCCACGGCGTCGACGGAGTTGTCGCTGTCGCCGCCGCCGGCGCAGGCGGCGGGTGGGGGAGGTGGTGGTGGTGGAGCGGACGCGCTGCCGTGGCGGCACCGACCTTCGCCGCCGTCGTCGGCGGTGGCCACCACGTCTGCCGCCGCCGCGGCGGCGCTGATGGCGCCGATGATGCTGCAGCCGCTCGACGCCGGCGGCGGCGCGTCGGCGCCGCCGCCGCCGATCCGCGGGATACCTATCTACAACGGCCCCGGCGGGTTCCCGTTCCTGCAGCCGTCGCCCACCGCCGGCGACGTCGGCCACCACCACCACCACCACCCCAAGATGGGATTCTACAGCTCGTACCACCACCCATCCACGTGGCCCTCCACGTCGCCGTCCCCGCTCGCGGCGCCGCCGGGCGCCGCGTCGTCGCCGCTCGACCCCACGGCGGCGTTCCTCTCCTCCCCCCACCACCGGATGCTGTCCGCCGCCTCGGGGAGGCTCAACGGCATGCTCTCCGTCTCCGACACCCTCCGCAGCTACGGCGTCCCCGGCGCCGCCGCCCCCGGCGTCATCGGCGGCGCGCACCACCACCACCACCACCTCCACGGCGGCCAGCCGTTCGTCGGCGCCCTCGCGTCCCGCTTCATGCCCAAGCTCCCCGCCAAGCGCAGCATGCGCGCGCCGCGCATGCGCTGGACGAGCACCCTCCACGCCCGCTTCGTCCACGCCGTCGAGCTCCTCGGCGGCCACGAGAGGGCGACGCCCAAGTCGGTGCTGGAGCTCATGGACGTCAAGGATCTGACGCTAGCGCATGTCAAGAGCCACCTCCAGATGTATCGCACCGTGAAGAGCACTGACAAGCCTGCAGCCTCTTCAGGGCCGGCGGACGGCGGCTCCGGCGACGAGGAGTTCGCCGGCGGCGGGCAGGCGGCGTCGGGCGGCGGCGACAGCATGTGCCTGAGGGGTGGCGGCGGCGGCGGGGTGGCCGCGGCGGCGTTCGCGGAGCACGGCCGGTCGGCGTCGGAGGGCGCCGCCAGCTCGGTCGGCGGCGGCGGCGGCGGCGACATGGACCAGTCGTCGGCCGGCAACACCAGCACCACCAGGTGGAGCAACTCCTCAAGGGACCCATGGCTGTCGTCCAATTCTTGCAACATGGACGCCCATCGCTCCGTAGGATTGTCTTCTCCTATTGAGAACTTGGAACCGTGCAGATCGAGCAGCTCGCAGGTGTCCAACCATGAGCTGAGTAGCCCTAGTCTCGAGTTCACTCTAGGGAGGCCTGACTGGCACGGTGCAGATCATGATTAG
CDS Sequence
- >LOC_Os09g23200.1
ATGGCAATGGTGCGGGAGCTCGAGTTGATGACGAGCTGGAGCAACAGCATGGGGCGACACCGCTACCCAACGCGCATCCTCGTCGATTCCTTCGGGCATAAGTGCAGTGCCTCTGACAAGGGTGTGTGGACCTCCTGCAGCATTCGCGCACCCTTGCAAGGACGCGGCTCCTTCCGACGTGGCGCGAATATTCGCTTTGGGAGCTTGCCGAGCAGCGCGGCGGTGGCGACGTCGGGGGGAGGGCGTGGCGGCGGCGGGGTGGTGGTGGGAGGAGGAGGAGGGGACCCGTGGCGGAGGCTGGATGGGTCCACGGCGTCGACGGAGTTGTCGCTGTCGCCGCCGCCGGCGCAGGCGGCGGGTGGGGGAGGTGGTGGTGGTGGAGCGGACGCGCTGCCGTGGCGGCACCGACCTTCGCCGCCGTCGTCGGCGGTGGCCACCACGTCTGCCGCCGCCGCGGCGGCGCTGATGGCGCCGATGATGCTGCAGCCGCTCGACGCCGGCGGCGGCGCGTCGGCGCCGCCGCCGCCGATCCGCGGGATACCTATCTACAACGGCCCCGGCGGGTTCCCGTTCCTGCAGCCGTCGCCCACCGCCGGCGACGTCGGCCACCACCACCACCACCACCCCAAGATGGGATTCTACAGCTCGTACCACCACCCATCCACGTGGCCCTCCACGTCGCCGTCCCCGCTCGCGGCGCCGCCGGGCGCCGCGTCGTCGCCGCTCGACCCCACGGCGGCGTTCCTCTCCTCCCCCCACCACCGGATGCTGTCCGCCGCCTCGGGGAGGCTCAACGGCATGCTCTCCGTCTCCGACACCCTCCGCAGCTACGGCGTCCCCGGCGCCGCCGCCCCCGGCGTCATCGGCGGCGCGCACCACCACCACCACCACCTCCACGGCGGCCAGCCGTTCGTCGGCGCCCTCGCGTCCCGCTTCATGCCCAAGCTCCCCGCCAAGCGCAGCATGCGCGCGCCGCGCATGCGCTGGACGAGCACCCTCCACGCCCGCTTCGTCCACGCCGTCGAGCTCCTCGGCGGCCACGAGAGGGCGACGCCCAAGTCGGTGCTGGAGCTCATGGACGTCAAGGATCTGACGCTAGCGCATGTCAAGAGCCACCTCCAGATGTATCGCACCGTGAAGAGCACTGACAAGCCTGCAGCCTCTTCAGGGCCGGCGGACGGCGGCTCCGGCGACGAGGAGTTCGCCGGCGGCGGGCAGGCGGCGTCGGGCGGCGGCGACAGCATGTGCCTGAGGGGTGGCGGCGGCGGCGGGGTGGCCGCGGCGGCGTTCGCGGAGCACGGCCGGTCGGCGTCGGAGGGCGCCGCCAGCTCGGTCGGCGGCGGCGGCGGCGGCGACATGGACCAGTCGTCGGCCGGCAACACCAGCACCACCAGGTGGAGCAACTCCTCAAGGGACCCATGGCTGTCGTCCAATTCTTGCAACATGGACGCCCATCGCTCCGTAGGATTGTCTTCTCCTATTGAGAACTTGGAACCGTGCAGATCGAGCAGCTCGCAGGTGTCCAACCATGAGCTGAGTAGCCCTAGTCTCGAGTTCACTCTAGGGAGGCCTGACTGGCACGGTGCAGATCATGATTAG
Protein Sequence
- >LOC_Os09g23200.1
MAMVRELELMTSWSNSMGRHRYPTRILVDSFGHKCSASDKGVWTSCSIRAPLQGRGSFRRGANIRFGSLPSSAAVATSGGGRGGGGVVVGGGGGDPWRRLDGSTASTELSLSPPPAQAAGGGGGGGGADALPWRHRPSPPSSAVATTSAAAAAALMAPMMLQPLDAGGGASAPPPPIRGIPIYNGPGGFPFLQPSPTAGDVGHHHHHHPKMGFYSSYHHPSTWPSTSPSPLAAPPGAASSPLDPTAAFLSSPHHRMLSAASGRLNGMLSVSDTLRSYGVPGAAAPGVIGGAHHHHHHLHGGQPFVGALASRFMPKLPAKRSMRAPRMRWTSTLHARFVHAVELLGGHERATPKSVLELMDVKDLTLAHVKSHLQMYRTVKSTDKPAASSGPADGGSGDEEFAGGGQAASGGGDSMCLRGGGGGGVAAAAFAEHGRSASEGAASSVGGGGGGDMDQSSAGNTSTTRWSNSSRDPWLSSNSCNMDAHRSVGLSSPIENLEPCRSSSSQVSNHELSSPSLEFTLGRPDWHGADHD*